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In scientific studies of other receptor tyrosine kinases implicated from the oncogenesis of GIST, nilotinib attained strong and selective inhibition of PDGFRα and PDGFRβ. As is the situation with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells transformed by PDGFRADrink lots of fluids all through treatment using this t
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